Surgical Planning Laboratory - Brigham & Women's Hospital - Boston, Massachusetts USA - a teaching affiliate of Harvard Medical School

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Figure 1: MR Images of Large Cavum Septi Pellucidi in Schizophrenic Patients (left panel) and No Cavum Septi Pellucidi in Normal Subjects (right panel).
Figure 1: MR Images of Large Cavum Septi Pellucidi in Schizophrenic Patients (left panel) and No Cavum Septi Pellucidi in Normal Subjects (right panel).
Figure 1: Upper Left: Intraoperative photograph showing the exposed tumor following opening of the dura mater. The tip of the probe is shown on the left, pointing to the area that caused speech arrest during cortical stimulation. Upper Right: Three-dimensional reconstructed image of the patient's brain. The vessels are represented in blue, the ventricles in light blue, the precentral gyrus in purple, and the tumor in green. The red dot corresponds to the speech area. The white arrow represents the probe on the adjacent intraoperative image. Lower: Orthogonal MR slices and cross-hairs representing the probe position.
Figure 1: Upper Left: Intraoperative photograph showing the exposed tumor following opening of the dura mater. The tip of the probe is shown on the left, pointing to the area that caused speech arrest during cortical stimulation. Upper Right: Three-dimensional reconstructed image of the patient's brain. The vessels are represented in blue, the ventricles in light blue, the precentral gyrus in purple, and the tumor in green. The red dot corresponds to the speech area. The white arrow represents the probe on the adjacent intraoperative image. Lower: Orthogonal MR slices and cross-hairs representing the probe position.
Figure 6: Images from a case report on 76 yo female, glioma in the posterior left frontal lobe. On the left: T1-w FSE image subtraction. On the right: water proton chemical shift imaging.
Figure 6: Images from a case report on 76 yo female, glioma in the posterior left frontal lobe. On the left: T1-w FSE image subtraction. On the right: water proton chemical shift imaging.
Figure 1: a) T1-weighted MR-image (SPGR), coronal plane in a preterm infant (31 wks) b) T2-weighted MR-image reformatted from axial to coronal plane and registered with SPGR image. c) Final segmented image depicting cortical GM in gray, unmyelinated WM in red, CSF in blue, myelinated WM in green and subcortical GM (basal ganglia) in white.
Figure 1: a) T1-weighted MR-image (SPGR), coronal plane in a preterm infant (31 wks) b) T2-weighted MR-image reformatted from axial to coronal plane and registered with SPGR image. c) Final segmented image depicting cortical GM in gray, unmyelinated WM in red, CSF in blue, myelinated WM in green and subcortical GM (basal ganglia) in white.
Figure 2: (a) Fourier encoded T,-weighted medial sagittal brain image (256 X 192, 4.0 mm thick) of a GA 31 -week infant, including the infant's hand, representing the FOV of simulation Case II; (b) Fourier encoded T, -weighted 256x192 lateral sagittal brain image (256 x 256, 4.0-mm thick) shifted 5.0 mm from the slice shown in (a), used to compute the SVD spatial encodes (Note: the absence of the infant's hand); (c) SVD encoded MRI simulated results obtained using 32 vertical and 32 horizontal SVD encodes; (d) SVD encoded MRI simulated results obtained using 64 vertical and 64 horizontal SVD encodes.
Figure 2: (a) Fourier encoded T,-weighted medial sagittal brain image (256 X 192, 4.0 mm thick) of a GA 31 -week infant, including the infant's hand, representing the FOV of simulation Case II; (b) Fourier encoded T, -weighted 256x192 lateral sagittal brain image (256 x 256, 4.0-mm thick) shifted 5.0 mm from the slice shown in (a), used to compute the SVD spatial encodes (Note: the absence of the infant's hand); (c) SVD encoded MRI simulated results obtained using 32 vertical and 32 horizontal SVD encodes; (d) SVD encoded MRI simulated results obtained using 64 vertical and 64 horizontal SVD encodes.
Figure 3: 3D model of the renal lesion and surrounding structures displayed using the LAVA tool.
Figure 3: 3D model of the renal lesion and surrounding structures displayed using the LAVA tool.
Figure 3: Reconstructions of the cerebral ventricles created from MR images. (a) Normal, healthy adult. (b) Patient with Alzheimer's disease. (c) Patient with normal-pressure hydrocephalus.
Figure 3: Reconstructions of the cerebral ventricles created from MR images. (a) Normal, healthy adult. (b) Patient with Alzheimer's disease. (c) Patient with normal-pressure hydrocephalus.
Figure 5: Model generation. a: Projection of skin vertices; b: skin–bone connections; c: mass-spring mesh.
Figure 5: Model generation. a: Projection of skin vertices; b: skin–bone connections; c: mass-spring mesh.
Figure 6: Cystic lesion next to the pancreatic duct generated together with the pancreatic duct. Pillarlike septa between the pancreatic duct and the cystic lesion were demonstrated in some cases.
Figure 6: Cystic lesion next to the pancreatic duct generated together with the pancreatic duct. Pillarlike septa between the pancreatic duct and the cystic lesion were demonstrated in some cases.
Figure 2: Image display on our implentation which consists of four windows. Three of these windows show coronal (A), sagittal (B), and axial (C) views of 2D gray scale images, overlaid by different color components from connected-component analysis inside the VOI (the rectangular box with color coding scheme on each 2D image displays). The fourth window (D) shows 3D-rendered image from connected-component analysis.
Figure 2: Image display on our implentation which consists of four windows. Three of these windows show coronal (A), sagittal (B), and axial (C) views of 2D gray scale images, overlaid by different color components from connected-component analysis inside the VOI (the rectangular box with color coding scheme on each 2D image displays). The fourth window (D) shows 3D-rendered image from connected-component analysis.
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